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magnetonin.com

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Welcome to IPRS's Magnesium Project - Magnetonin Discovery
www.magnetonin.com

What is Magnetonin(tm)?
Magnetonin is the name given to the polypeptide hormone that is presumed (at this point in time) to be responsible for the regulation of magnesium metabolism.

What is the Origin of Magnetonin Concept?
The Magnetonin Concept has come about as a result of research being carried out as part of the Mg Project of IPRS, Inc.

What is the evidence for Magnetonin?
All other major electrolytes of the body are regulated by one or more regulatory mechanisms including but not limited to a hormone that when released elevates the level of the cation in the blood and in selected tissues/organs. Given the importance of Mg levels in the blood and many different tissues, it seems highly improbable that there is not also a hormone to regulate it.

What will it do?
It should be active on promoting uptake from the gut, reabsorption from the kidneys and mobilization from bone. It may also activate Mg-channels in muscle and neural tissues where Mg is especially vital to normal functioning.

Where is it Produced?
If similar to calcitonin, it will likely be found in more concentrated form in the parathyroid glands.

Objectives
Licensable technology for commercial development for treating many diseases.

Magnetonin Initiative Implementation
The search of Magnetonin is being carried out at many levels and is expected to proceed in many institutions and research laboratories throughout the world.

One of the steps in this discovery process is in defining the likely attributes of magnetonin. One assumption is that magnetonin will have similar attributes to other electrolyte regulators and especially to calcitonin. A matrix has been developed to frame these discussions.

A second step is to compare the physical/chemical properties of the electrolyte regulated. A comparison with other electrolytes in the body should be instructive.

A third step is to look at the properties of the "sister" hormones, calcitonin and parathyroid hormone. The details of their functioning and disorders associated with them are being developed.

A fourth step is to develop/adapt laboratory techniques for measuring the presence of the novel substance(s). Click here for suggestions.

Other steps are to share data across laboratories and develop tools for analysis both for laboratory studies and clinical studies. Provide a forum for information and idea exchange. Identify scientists and research institutions, publications and web sites.

Lesson #1
Look for elevated levels of serum/plasma magnesium when tumors, hyperplasias or cancers are found in the parathyroids, thyroid and adrenal glands. Conversely, look for decreased levels of magnesium when any of these organs are compromised by surgical or chemical means.

Lesson #2
Look for parallels between hyper- and hypo- calcemia symptoms and hyper/hypo-magnesemia; namely, for hypo-magnesemia look for tetany, circumoral paresthesia, tonic contractions in hands, feet, face, eyes, tongue and larynx; and for hyper-magnesemia look for weakness, anorexia, nausea, constipation, ileus, abdominal pain, nocturia, polyuria, and thirst.

Lesson #3
Based on the smaller size of magnesium ion compared to calcium and what we know to date of bone formation, we are inclined to hypothesize that magesium is less involved in bone formation and that therefore magnetonin is less like calcitonin and more like the parathyroid hormone. That is to say, magnetonin will likely have more influence on membrane transport, nerve and muscle excitability, protein activity and membrane stability than on bone.

Other approaches and details can be obtained from IPRS.


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